Dr L. Simoncini -- Sodium Bicarbonate Therapy vs Cancer
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**Dr Tullio
SIMONCINI**
**Sodium
Bicarbonate vs Candida & Cancer**
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**SIMONCINI CANCER THERAPY
-- DR. TULLIO SIMONCINI WRITES**
My idea is that cancer doesnt depend on mysterious
causes (genetic, immunological or auto immunological as the
official oncology proposes), but it comes down from a simple
fungal infection, whose destroying power in the deep tissues is
actually under estimated.
**Premise**
The present work is based on the conviction, supported
by many years of observations, comparisons and experiences, that
the necessary and sufficient cause of the tumour is to be sought
in the vast world of the fungi, the most adaptable, aggressive and
evolved micro-organisms known in nature.
I have tried many times to explain this theory to
leading institutions involved in cancer issues (the Ministry of
Health, the Italian Medical Oncological Association, etc.)
elaborating on my thinking, but I have been brushed aside because
of the impossibility of setting my idea in a conventional context.
A different, international audience represents the
possibility of sharing a view about health, which differs, from
what is widely accepted by today's medical community, either
officially or from the sidelines.
There is an opposition between the allopathic and the
Hippocratic medical ideal. The former has the disadvantage of its
inability to consider the individual as a whole. Therefore it
brings with it all the distortions and aberrations which such a
point of view entails (excessive specialisation, therapeutic
aggressiveness, superficiality, harmfulness etc.). The latter
approach instead tends in the direction of being too generic,
non-scientific, and devoid of therapeutic incisiveness.
The position that I promote represents instead a meeting
point of these two conceptions of health, since, from the
conceptual point of view, it sublimates and adds value to both,
while highlighting how they both are victims of a common
conformist language.
The hypothesis of a fungal aetiology in
chronic-degenerative illness, able to connect the ethical
qualities of the individual with the development of specific
pathologies, reconciles the two orientations (allopathic and
holistic) of medicine. The hypothesis is a strong candidate for
being that missing element of psychosomatics that has been sought
but never found by one of the fathers of psychosomatics, Wiktor
Von Weiszacker.
In considering the biological dimensions of the fungi,
for instance, it is possible to compare the different degrees of
pathogenicity in relation to the condition of organs, tissues and
cells of a guest organism, which in turn also and especially
depend on the behaviour of the individual.
Each time the recuperative abilities of a known
psycho-physic structure are exceeded, there is an inevitable
exposure, even considering possible accidental cofounders, to the
aggression -- even at the smallest dimensions -- of those external
agents that otherwise would be harmless.
In the presence of an indubitable connection between
patient morale and disease it is no longer legitimate to separate
the two domains (allopathic and naturopathic) which are both
indispensable for improving the health of individuals.
The Platonic separation of the human mind from the human
body, responsible for the present mechanistic and materialistic
character of today's medicine, is outdated. So is the pessimistic
Kantian position concerning integration of the rational and
emotional sides of man ("the starred sky above me, the moral law
within me"), which generates the present myopia of today's medical
epistemology. With such outdated cognitive frameworks inevitably
come all the mindsets that carry similar restrictive and limiting
presuppositions.
**Candida Albicans: Necessary and Sufficient Cause of
Cancer**
When facing the most pressing contemporary medical
problem, cancer, the first thing to do is to admit that we still
do not know its real cause. However treated in different ways by
both official and alternative medicine, an aural of mystery still
exists around its real generative process.
The attempt to overcome the present impasse must
therefore and necessarily go through two separate phases: a
critical one that exposes the present limitations of oncology, and
a constructive one capable of proposing a therapeutic system based
on a new theoretical point of departure.
In agreement with the most recent formulation of
scientific philosophy, which suggests a counter-inductive approach
where it is impossible to find a solution with the conceptual
tools that are commonly accepted, only one logical formulation
emerges; that is, to refuse the oncological principle which
assumes cancer is generated by a cellular reproductive anomaly.
However, if the fundamental hypothesis of cellular
reproductive anomaly is questioned, it becomes clear that all the
theories based on this hypothesis are inevitably flawed.
It follows that both an auto-immunological process, in
which the body's defence mechanisms against external agents turn
their destructive capacity against internal constituents of the
body, and an anomaly of the genetic structure implicated in the
development of auto-destruction, are inevitably disqualified.
Moreover, the common attempt to construct theories about
multiple causes that have an oncogenic effect on cellular
reproduction sometimes seems like a concealing screen, behind
which there is nothing but a wall. These theories propose endless
causes that are more or less associated with each other; and this
means in reality that no valid causes are found. The invocation in
turn of smoking, alcohol, toxic substances, diet, stress,
psychological factors, etc., without a properly defined context,
causes confusion and resignation, and creates even more
mystification around a disease which may turn out to be simpler
than it is depicted to be.
As background information, it is important to review the
picture of presumed genetic influences in the development of
cancer processes as they are depicted by molecular biologists.
These are the scientists who perform research on infinitesimally
small cellular mechanisms, but who in real life never see a
patient. All present medical systems are based on this research,
and thus, unfortunately, all therapies currently performed.
The main hypothesis of a genetic neoplastic causality is
essentially reduced to the fact that the structures and the
mechanism in charge of normal reproductive cellular activity
become, for undefined causes, capable of an autonomous behaviour
that is disjointed from the overall tissular economy.
The genes that normally have a positive role in cellular
reproduction are, then, imprecisely referred to as
proto-oncogenes; those which inhibit cellular reproduction are
called suppressor genes or recessive oncogenes.
Both endogenous (never demonstrated) and exogenous
cellular factors -- that is, those carcinogenic elements that are
usually invoked -- are held responsible for the neoplastic
degeneration of the tissues.
In J.H. Stein (*Medicina Interna - Internal Medicine,*
Mosby Year Book inc.1994, St. Louis, Missouri, 4th edition,
Milano, 1995, page 1186 -1187) the following is reported:
The mitogenic signals, from the microenvironment or from
more distant areas of influence, are transmitted to the cells
through numerous receptive structures that are associated to the
plasmatic membrane.
Among these structures, the ones that have been studied
most exhaustively are receptors with an external domain for the
binder, a transmembranic domain and a cytoplasmatic domain with a
thyrosinkinase activity.
Besides these, it is thought that at least seven
distinct classes of molecules participate in the transmission of
the mutagenic signal:
1) receptors coupled to G proteins
2) ionic channels
3) receptors with intrinsic activity guanil cyclase
4) receptors for many lymphofokines, cytokines and
growth factors (interleukine, eritropoietine, etc.)
5) receptors for the phosphothyrosine phosphorilase
activity
6) nuclear receptors belonging to the supergenic family
of the receptor for steroidal estrogenic and thyroidal hormones
7) Finally, increasing numbers of tests suggest that the
adhesion molecules expressed on the surface of the cells
communicate with the microenvironment in ways that produce very
important consequences for cellular growth and differentiation.
From a very superficial analysis of this presumed
oncological picture, however, it seems to be clear how the
assertion of all this unstoppable genetic hyperactivity, generated
by elements that almost seem to lurk in the realms of the sinister
and the monstrous, and that therefore suggest the existence of
God-knows-what abysmal mechanisms that can only be deciphered with
equally abysmal conceptual mechanisms -- all this can do nothing
more that unveil the abysmal stupidity that is at the basis of
this way of conceiving things.
What is even more serious is the fact that nobody in the
present health establishment seems to question the above-mentioned
stupidities. All those who work in the field do nothing but repeat
the stale litany of reproductive cellular anomalies on a genetic
basis.
Since in this state of affairs the present medical
theory shows an impoverishment and a superficiality that are
indeed abysmal, it is better to look for new horizons and
conceptual instruments that are capable of unearthing a real and
unique neoplastic aetiology.
After so many years of failure and suffering it is time
to rejuvenate minds with new and productive juices. Arguments for
mysterious and complex genetic factors, a monstrous reproductive
capacity by a pathologic entity capable of tearing apart any
tissue, the idea that there is an implicit and ancestral tendency
of the human organism to deviate in an auto-destructive sense --
these and other similar arguments, spiced with exponentially
multiplying numbers of "ifs" and "maybes" -- it all has the
flavour more of raving free-association than of a healthy
scientific discourse.
Once the present oncological perspectives have been refuted,
however, it is legitimate to ask how the successes achieved by
official medicine and by alternative medicine have to be
classified.
To this end, it is useful to remember that contemporary
epistemology has demonstrated how the contributions to causality
of contextual and co-textual elements of a theory, if they cannot
be defined, are therefore chancy, especially in ultra-dimensional
space, that is, in the microscopic dimension.
In practical terms, this means that data or positive
facts that are considered proof when concerning a basic principle
(for example, the above-mentioned cellular reproductive
anomalies), and therefore obtained by utilising a limited number
of variables next to the complexity of human disease, cannot be
trusted, since they work only from the initial hypothetical
functions.
Where, in fact, we admit the possibility of improvements
or cures, it is not admissible from the logical point of view
attribute them to this or that method of cure that is more of less
official, since it is not possible to specify and include all or
the majority of the components that are at play in the object man,
in whom conditions of certainty cannot exist.
Paradoxically, the possible positive effect of each
therapeutic system could derive from elements that are not
foreseen and are unknown to all. Those elements, however, could be
influenced by or determined to some extent by one another.
We may find ourselves in a position in which everybody
rightfully has the right to promote his point of view, without
knowing the real reasons for his successes.
In this case, then, even the most rigorous
experimentation takes on a fictional character rather than the
function of a true correspondence with reality, and the end result
is a continuous sterile petitio principii.
If we then put aside completely the conceptual frame of
contemporary oncology with all its interpretative variables of
genetic, immuncological and toxicological character, what is left
as the only logical, practicable way is the domain of the
infectious diseases, to be seen and reconsidered with different
eyes that has been the case so far.
Two considerations support such a conclusion. One is of
a historical nature, and the other is of an epidemiological
nature. The former derives from the fact that, in the
therapeutical approach to the patient, the improvement in quality,
that is the possibility of a real cure for the patient, has been
determined almost exclusively by the development of microbiology.
The latter derives from the analysis of life expectancy that has
taken place in the last decades which, since it is associated with
an inevitable change of the sthenicity of individuals, can be
hypothesised as a determining factor in the development atypical
infectious pathologies.
In order to find the possible carcinogenic ens morbi on
the horizon of microbiology, it appears useful to return to the
basic taxonomical concepts of biology, where we can see,
incidentally, the existence of a noticeable amount of indecision
and indetermination.
Already in the last century, a German biologist, Ernest
Haeckele (1834-1919), departing from the Linnaeian concept that
makes for two great kingdoms of living things (vegetable and
animal) denounced the difficulties of categorising all those
microscopic organisms which, because of their characteristics and
properties, could not be attributed to either the vegetable or
animal kingdom. For these organisms, he proposed a third kingdom,
called Protists.
"This vast and complex world includes a range of
entities beginning with those that have sub-cellular structure --
existing at the limits of life -- such as viroids and viruses,
moving through the mycoplasms, to finally, organisms of greater
organisation: bacteria, actinomycetes, mixomycetes, fungi,
protozoa, and perhaps even some microscopic algae." (2).
The common element of these organisms is the feeding
system, which, being implemented (with very few exceptions) by
direct absorption of soluble organic compounds, differentiates
them both from animals and vegetables. Animals also feed as above,
but especially by ingesting solid organic materials that are then
transformed through the digestive process. Vegetables are capable,
by utilising mineral compounds and light energy, to feed by
synthesising the organic substances.
The contemporary tendency of biologists is to once again
pick up, though in a more sophisticated way, the concept of the
third kingdom. One goes even further, however, arguing that within
that kingdom, fungi must be classified in a distinct category.
O. Verona (3) says that if we put multicellular
organisms provided with photosynthetic capabilities (plants) in
the first kingdom, and the organisms not provided with
photosynthetic pigmentation (animals) in the second kingdom, and
organisms from both these kingdoms are made of cells provided with
a distinct nucleus (eukaryotes); and, furthermore, if we put in
another kingdom (protists) those monocellular organisms that have
no chlorophyll and have cells that are without a distinct nucleus
(prokaryotes), the fungi can well have their own kingdom because
of the absence of photosynthetic pigmentation, the ability to be
mono-cellular, and multi-cellular, and, finally, their possession
of a distinct nucleus.
Additionally, fungi possess a property that is strange
when compared to all other micro-organisms: the ability to have a
basic microscopic structure (hypha) with a simultaneous tendency
to grow to remarkable dimensions (up to several kilograms),
keeping unchanged the capacity to adapt and reproduce at any size.
From this point of view, therefore, fungi cannot be
considered true organisms, but cellular aggregates sui generis
with an organismic behaviour, since each cell maintains its
survival and reproductive potential intact regardless of the
structure in which it exists.
It is therefore clear how difficult it is to identify
all the biological processes in such complex living realities. In
fact, even today, there are huge voids and taxonomical
approximations in mycology.
It is worthwhile to examine more deeply this strange world, with
such peculiar characteristics, and try to highlight those elements
that somehow may be pertinent to the problems of oncology.
1) Fungi are heterotrophic organisms and therefore need,
as far as nitrogen and carbon are concerned, pre-formed compounds.
Of these compounds, simple carbohydrates, for example
monosaccarides (glucose, fructose, and mannose) are among the most
utilised sugars. This means that fungi, during their life cycle,
depend on other living beings, which must be exploited in
different degrees for their feeding. This occurs both in a
saprophytic way (that is, by feeding on organic waste) and in a
parasitic way (that is, by attacking the tissue of the host
directly).
2) Fungi show a great variety of reproductive
manifestations (sexual, asexual, gemmation; these manifestations
can often be observed simultaneously in the same mycete), combined
with a great morphostructural variety of organs. All of this is
directed toward the end of spore formation, to which the
continuity and propagation of the species is entrusted.
3) In mycology, it is often possible to observe a
particular phenomenon called heterocarion, characterised by the
coexistence of normal and mutant nuclei in cells that have
undergone a hyphal fusion.
Nowadays, phitopathologists are quite worried about the
creation of individuals that are genetically quite different even
from the parents. This difference has taken place by means of
those reproductive cycles, which are called parasexual.
The indiscriminate use of phitopharmaceuticals has in
fact often determined mutations of the nuclei of many parasite
fungi with the consequent creation of heterocarion -- and this is
sometimes particularly virulent in its pathogenicity (4).
4) In the parasitic dimension, fungi can develop from
the hyphas more or less beak-shaped specialised structures that
allow the penetration of the host.
5) The production of spores can be so abundant as to
always include, at every cycle, tens, hundreds, and even thousands
of millions of elements that can be dispersed at a remarkable
distance from the point of origin (a small movement is sufficient,
for example, to implement immediate diffusion).
6) Spores have an immense resistance to external
aggression, for they are capable of staying dormant in adverse
conditions for many years, while preserving unaltered their
regenerative potentialities.
7) The development coefficient of the hyphal apexes
after the germination is extremely fast (100 microns per minute
under ideal conditions) with ramification capacity, thus with the
appearance of a new apex region that in some cases is in the
neighbourhood of 40-60 seconds (6).
8) The shape of the fungus is never defined, for it is
imposed by the environment in which the fungus develops.
It is possible to observe, for example, the same
mycelium in the simple isolated hyphas status in a liquid
environment or in the form of aggregates that are increasingly
solid and compact up to the formation of pseudoparenchymas and of
filaments and mycelial strings (7).
9) By the same token, it is possible to observe in
different fungi the same shape whenever they must adapt to the
same environment (this is called dimorphism).
The partial or total substitution of nourishing substances induces
frequent mutations in fungi, and this is further proof of their
high adaptability to any sub-strata.
10) When the nutritional conditions are precarious, many
fungi react with hyphal fusion (among nearby fungi) which allows
them to explore the available material more easily, using more
complete physiological processes.
This property, which substitutes co-operation for
competition, makes them distinct from any other microorganism, and
for this reason Buller calls them social organisms (8).
11) When a cell gets old or becomes damaged (i.e. by a
toxic substance or by a pharmaceutical) many fungi whose
intercellular septums are provided with a pore react by
implementing of a defence process called protoplasmic flux through
which they transfer the nucleus and cytoplasm of the damaged cell
into a healthy one, thus conserving unaltered all their biological
potential.
12) The phenomena regulating the development of hyphal
ramification are unknown to date (9). They consist either of a
rhythmic development, or in the appearance of sectors which,
though they originate from the hyphal system, are self-regulating
(10), that is, independent of the regulating action and behaviour
of the rest of the colony.
13) Fungi are capable of implementing an infinite number
of modifications to their own metabolism in order to overcome the
defence mechanism of the host. These modifications are implemented
through plasmatic and biochemical actions as well as by a
volumetric increase (hypertrophy) and numerical hyperplasy of the
cells that have been attacked (11).
14) Fungi are so aggressive as to attack not only
plants, animal tissue, food supplies and other fungi, but even
protozoa, amoebas and nematodes.
Fungi hunt nematodes, for example, with peculiar hyphal
modifications that constitute real mycelial criss-cross, viscose,
or ring traps that achieve the immobilisation of the worms, as a
precursor their hyphal invasion.
In some cases, the aggressive power of fungi is so great
as to allow it, with only a cellular ring made up of three units,
to tighten in its grip, capture and kill its prey in a short time
notwithstanding the prey's desperate struggling.
From the short notations above, therefore, it seems fair
to dedicate a greater attention to the world of fungi, especially
considering the fact that biologists and microbiologists
constantly highlight large deficiencies and voids in all their
descriptions and interpretations of the fungi's shape, physiology
and reproduction.
So the fungus, which is the most powerful and the most
organised micro-organism known, seems to be an extremely logical
candidate as a cause of neoplastic proliferation. Imperfect Fungi
(so called because of the lack of knowledge and understanding of
their biological processes) deserve particular attention since
their essential prerogative sits in their fermentative capacity.
The greatest disease of mankind may therefore hide
within the small cluster of pathogenic fungi, and may be after all
be located with just some simple deductions able to close the
circle and providing the solution.
Considering that, among the human parasite species, the
Dermatophytes and Sporotrichum demonstrate an excessively specific
morbidity, and that experience shows that Actinomycetes,
Toluropsis and Hystoplasma rarely enter the context of pathology,
the Candid Albicans clearly emerges as the sole candidate for
tumoral proliferation.
If we stop for a second and reflect on its
characteristics, we can observe many analogies with neoplastic
disease. The most evident are:
1) Ubiquitous attachment: no organ or tissue is spared
2) The constant absence of hyperpyrexia
3) Sporadic and indirect involvement of the differential
tissues
4) Invasiveness that is almost exclusively of the focal
type
5) Progressive debilitation
6) Refractivity to any type of treatment
7) Proliferation facilitated by multiplicity of
indifferent cofounders
8) Symptomatological basic configuration with structure
tending to the chronic
Therefore an exceptionally high and diversified
pathogenic potentiality exists in this mycete of just a few
microns in size, which, even though it cannot be traced with the
present experimental instruments, cannot be neglected from the
clinical point of view.
Certainly, its present nosological classification cannot
be satisfactory, because if we do not keep the possibly endless
parasitic configurations in mind, that classification is too
simplistic and constraining.
We therefore have to hypothesise that Candida, in the
moment it is attacked by the immunological system of the host or
by a conventional antimycotic treatment, does not react in the
usual, predicted way, but defends itself by transforming itself
into ever-smaller and non-differentiated elements that maintain
their fecundity intact to the point of hiding their presence both
to the host organism and to possible diagnostic investigations.
The Candida's behaviour may be considered to be almost
elastic:
When favourable conditions exist, it thrives on an
epithelium; as soon as the tissue reaction is engaged, it
massively transforms itself into a form that is less productive
but impervious to attack -- the spore.
If then continuous sub-epithelial solutions take place
coupled with a greater a-reactivity in that very moment the spore
gets deeper in the lower connective tissue in such an impervious
state, it is irreversible.
In fact, the Candida takes advantage of a structural
interchangeability utilising, according to the difficulties to
overcome its biological niche.
In this way, Candida is free to expand to maturation in
the soil, air, water, vegetation, etc., that is, wherever there is
no antibody reaction.
In the epithelium, instead, it takes a mixed form, that
is reduced to the sole spore component when it penetrates in the
lower epithelial levels, where it tends to expand again in the
presence of conditions tissular a-reactivity.
The initial mandatory step of an in-depth research
endeavour would be to understand if and in which dimensions the
spore transcends; what mechanisms it engages to hide itself or,
again, if it preserves its parasitical characteristic, or if it
has available a neutral quiescent position, which is difficult or
even impossible to detect by the immunological system.
Unfortunately today we do not have the appropriate
means, either theoretical or technical, to answer these and
similar questions, so that the only valid suggestions can come
solely from clinical observation and experience. While not
providing immediate solutions, these sources can at stimulate
further questions.
Assuming that Candida Albicans is the agent responsible
for tumoral development, a targeted therapy would keep into
account not just its static and macroscopic manifestations, but
even the ultramicroscopic ones especially in their dynamic
valence, that is, the reproductive.
It is very probable that the targets to attack are the
fungi's dimensional transition points in order to perform a
decontamination with such a scope as to include the whole spectrum
of the biological expression: parasitic, vegetative, sporal, and
even ultra-dimensional and, to the limit, viral.
If we stop at the most evident phenomena, we risk
administering salves and unguents forever (in the case of
dermatomycosis or in psoriasis), or to clumsily attack (with
surgery, radiotherapy or chemotherapy) enigmatic tumoral masses
with the sole result of facilitating their propagation, which is
already heightened in the mycelial forms.
Why, one may ask, should we assume a different and
heightened activity of Candida Albicans since it has been
abundantly described in its pathological manifestations?
The answer lies in the fact that it has been studied
only in a pathogenic context, that is, only in relation to the
epithelial tissues. In reality Candida possesses an aggressive
valence that is diversified in function of the target tissue. It
is just in the connective or in the connective environment, in
fact, and not in the differentiated tissues, that Candida may find
conditions favourable to an unlimited expansion.
This emerges if we stop and reflect for a moment on the
main function of connective tissue, which is to convey and supply
nourishing substances to the cells of the whole organism.
This is to be considered as an environment external to
the more differentiated cells such as nervous, muscular, etc. It
is in this context, in fact, that the alimentary competition takes
place.
On one hand we have the organism's cellular elements
trying to defeat all forms of invasion; on the other hand, we have
fungal cells trying to absorb ever-growing quantities of
nourishing substances, for they have to obey the species'
biological imperative to form ever-larger and diffused masses and
colonies.
From the combination of various factors pertinent both
to the host and the aggressor, it is possible to hypothesise the
evolution of a candidosis;
First stage Integer epitheliums, absence of the
debilitating factors. Candida can only exist as saprophyte
Second Non-integer epitheliums (erosions, abrasions,
etc.), absence of stage debilitating factors, unusual transitory
conditions (acidosis, metabolic disorder, and microbial disorder).
Candida expands superficially (classic mycosis, both exogenous and
endogenous).
Third Non-integer epitheliums, presence of debilitating
factors (toxic, stage radiant, traumatic, neuropsychic, etc.).
Candida goes deeper into the sub-epithelial levels from which it
can be carried to the whole organism through the blood and lymph
(intimate mycosis). (12)
Stages one and two are the most studied and known, while
stage three, though it has been described in its morphological
diversity, is reduced to a silent form of saprophytism.
This is not acceptable from a logical point of view,
because no one can demonstrate the harmlessness of the fungal
cells in the deepest parts of the organism.
In fact, the assumption that Candida can behave in the
same saprophytic manner that is observed on integer epitheliums
when it has successfully penetrated the lower levels is at least
risky, because the assumption would have to be sustained by
concepts that are totally aleatory.
In fact, we asked not only to accept a priori that the
connective environment is (a) not suitable to nourish the Candida,
but also at the same time to accept (b) the omnipotence of the
body's defence system towards an organic structure that is
invasive but that then becomes vulnerable once lodged in the
deeper tissues.
As to point a), it is difficult to imagine that a
micro-organism so able to adapt itself to any sub-strata cannot
find elements to support itself in the human organic substance; by
the same token, it seems risky to hypothesise that the human
organism's defence system is totally efficient at every moment of
its existence.
Finally, the assumption that there is a tendency to a
state of quiescence and vulnerability in the case of a pathogenic
agent such as fungus -- the most invasive and aggressive
microorganism existing in nature -- seems to carry a whiff of
irresponsible.
It is therefore urgent, on the basis of the
above-mentioned considerations, to recognise the hazardous nature
of such a pathogenic agent, which is capable of easily taking the
most various biological configurations, both biochemical and
structural, in function of the condition of the host organism.
The fungal expansion gradient in fact becomes steeper as
the tissue that is the host of the mycotic invasion becomes less
eutrophic, and thus less reactive.
To that end, it seems useful to briefly consider the
"benign tumour" nosological entity. This is an issue that always
appears in general pathology but that indeed is brushed aside most
of the time too easily, and it is overlooked, since it usually
doesn't create either problems or worries. It constitutes one of
those underestimated grey areas seldom subjected to rational,
fresh consideration.
If the benign tumour, however, is not considered a full-fledged
tumour, it would be advantageous, for clarity, to categorise it in
an appropriate nosological scheme. If, instead, it is thought that
it fully belongs to neoplastic pathology, then it is necessary to
consider its non-invasive character and consequently to consider
the reasons for this.
It is in fact evident how in this second scenario, the
thesis based on a presumed predisposition of the organism to
auto-phagocytosis, having to admit an expressive graduation, would
stumble into such additional difficulties such as to become
extremely improbable.
By contrast, in the fungal scenario, the mystery of why
there are benign and malignant tumours is exhaustively solved,
since they can be recognised as having same etiological genesis.
The benignity or malignancy of a cancer in fact depends
on the capability of tissular reaction of a specific organ
expressing itself ultimately in the ability to encyst fungal
cells, and to prevent them from developing in ever-larger
colonies. This can be achieved more easily where the ratio between
differentiated cells and connective tissue is in favour of the
former.
Situated between the impervious noble tissues, then, and
the defenceless connective, the differentiated connective
structures (the glandular structures in particular) represent that
medium term which is only somewhat vulnerable to attack, because
of an ability to offer a certain type of defence.
And it is in these conditions that benign tumours are
formed, that is, where the glandular connective tissue is
successful in forming hypertrophic and hyperplastic cellular
embankments against the parasites.
In the stomach and in the lung, instead, since there are
no specific glandular units, the target organ, provided with a
small defensive capability, is at the mercy of the invader.
Furthermore, it is worth mentioning how several types of intimate
fungal invasion do not determine the appearance of malignant or
benign tumours, but a type of particular benign tumour (specific
degenerative alterations) as is the case of some organs or
apparatuses that do not have peculiar glandular structures, but
nevertheless are attacked in their connective tissue, but in a
limited way.
If we consider, in fact, multiple sclerosis, SLA,
psoriasis, nodular panartherite, etc. the possible development of
the fungus in a three-dimensional sense is actually limited by the
anatomic configuration of the invaded tissues, so that only a
longitudinal expansion is allowed.
Going back to the precondition of a-reactivity that is
necessary for neoplastic development in a specific individual, it
is permissible to affirm how in the human body each external or
internal element that determines a reduction of well-being in an
organism, organ or tissue, possesses oncogenic potentiality. This
is not so much because of an intrinsic damaging capability as much
as for a generic property of favouring the fungal (that is,
tumoral) flourishing.
Then the causal network so much invoked in contemporary
oncology, which involves toxic, genetic, immunological,
psychological, geographical, moral, social, and other factors,
finds a correct classification only in a mycotic infectious
perspective where the arithmetical and diachronic summation of
harmful elements works as a cofactor to the external aggression.
Having theoretically demonstrated the equivalency tumour
= fungus, it is clear how this interpretative key offers a long
series of questions concerning the contemporary therapies both
oncological (used without reference indexes) and antimycotic
(utilised only at a superficial level).
Which path is best to walk today, then, when faced with
a cancer patient, since the conventional oncological treatment,
not being etiological, can only occasionally have positive effects
and most of the time produces damage?
In the fungal perspective in fact, the effectiveness of
surgery is noticeably reduced because of the extreme diffusibility
and invasiveness characteristic of a mycelial conglomerate.
Surgery's to solve the problem is therefore tied to the case -- to
conditions, that is, in which one has the luck to completely
remove the entire colony (which is often possible in the presence
of a sufficient encystment; but here we are in the case of benign
tumours).
Chemotherapy and radiotherapy produce almost exclusively
negative effects, both for their specific ineffectiveness, and for
their high toxicity and harmfulness to the tissues, which in the
last analysis favours mycotic aggressiveness.
By contrast, an anti-fungal, anti-tumour specific
therapy would keep into account the importance of the connective
tissue, together with the reproductive complexity of fungi. Only
by attacking the fungi across the spectrum of all its forms, at
points where it is most vulnerable from the nutritional point of
view, would it be possible to hope to eradicate them from the
human organism.
The first step to take, therefore, would be to reinforce
the cancer patient with generic reconstituent measures (nutrition,
tonics, regulation of rhythms and vital functions), that are able
to enhance, by themselves, the general defences of the organism.
Concerning the possibility of having available
pharmaceutical cures which unfortunately do not exist today, it
seems useful, in the attempt to find an anti-fungal substance that
is quite diffusible and therefore effective, to consider the
extreme sensitivity of Candida towards sodium bicarbonate (i.e. in
the oral candidosis of breasted babies). This is consistent with
the fact that Candida has an accentuated ability to reproduce in
an acid environment.
Theoretically, therefore, if treatments that put the
fungus in direct contact with high bicarbonate concentrations
could be found, we should be able to see a regression of the
tumoral masses.
And this is what happens in many types of tumour, such
as colon, liver -- and especially stomach and lung -- the former
susceptible to regression just because of its "external" anatomic
position, the latter because of the high diffusibility of sodium
bicarbonate in the bronchial system and for its high
responsiveness to general reconstituent measures.
By applying a similar therapeutic approach, it has been
possible in some patients (about 30 in the last 15 years) to
achieve complete remission of the symptomatology and normalisation
of the instrumental data.
Following are the reports of seven cases of patients,
some of whom survived more than 10 years.
It is important to emphasise that these cases are
presented just as an example of what could be a new way of
perceiving the complexity of medical problems, especially in
oncology.
It is clear, in fact, that because of the very limited
number of cases, the lack of documentation showing rigid, orthodox
experimental methodology, and the long time that has elapsed since
these cases were treated, that the evidence required for strong
support of this theory on cancer is lacking.
I will not indicate in this paper the personal, cultural
and professional reasons that were responsible for the
interruption of the study and cure of cancer patients until
recently (that is, until two or three years ago, when I resumed
the treatment of cancer cases). I am however convinced that the
important fact that some patients have been able to heal and
survive for several years with therapies that are different from
the common, deadly therapeutic methods, must be divulged. This is
especially because these results come from a new way of thinking
which, as opposed to groping in the dark as official and various
alternative medicines do, has a well-defined subject -- fungi --
in a theory which of course is still to be proven and validated.
One may ask why more recent cases are not shown below.
This is because insufficient time has elapsed since treatment for
a demonstration of long-term well being of the patients, and
therefore these cases are not included.
It is also important to highlight that nowadays it is
very difficult to have a large number of cases, since it is not
easy to obtain a large number of cancer patients -- they are
addressed by the current system almost exclusively toward the
official channels of medicine, even if in many cases those have
been proven ineffective or deadly.
Keeping the above in mind, I consider it useful to
describe these cases as follows:
Case 1: A 70-year old female patient with diagnosis of
stomach adenocarcinoma confirmed by commonly accepted oncological
tests (TAK, biopsy, etc.). Two days before the scheduled
operation, she accepts the suggestion of trying a less sanguinary
approach, and leaves the hospital. For the period of a month, she
is administered sodium bicarbonate (one teaspoon in a glass of
water) to ingest half an hour before breakfast (that is, on an
empty stomach) for the purpose of maximising the effect. After
about two months normalisation of the gastric function takes place
with attenuation at first, and eventual loss of all the
symptomatology related to neoplastic pathology (lack of appetite,
digestion troubles, fatigue, lipothymic events, etc.). After an
endoscopic examination performed one year after the beginning of
therapy, the total remission of neoplastic formation is
ascertained and the patient refuses further investigation.
The patient is still alive today, 15 years after the treatment.
Case 2: A 67-year-old patient with a long history of
gastric ulcer is diagnosed with stomach cancer and a gastrectomy
is suggested. The patient, believing his disease is just an
exacerbation of the ulcer, wants to find an alternative to
surgery. He therefore accepted a therapy with sodium bicarbonate
as in case 1. The therapy determines in a few months the
regression of the neoplastic symptomatology. After about 18
months, during which no check-up is performed, upon the return of
symptomatology, treatment is resumed as above. Gastric
functionality is quickly re-established and maintained for about
eight years, after which contact with the patient is lost.
Case 3: A 58-year-old patient with stomach carcinoma is
diagnosed through histological examination performed on
endoscopical sample. The patient chooses not to undergo the
conventional therapies and he decides to accept a therapy similar
to that in the two preceding cases. The resulting effect is a
normalisation of symptomatology for about three years, that is,
until there are no further medical check-ups.
Case 4: In September, 1983, a 71-year-old patient
undergoes a hospital check-up in a serious condition of emaciation
caused by a large weight loss (about 15 Kgs.) which occurred over
the prior few months. Once a stomach neoplastic condition has been
diagnosed, and after the layout of a combined oncological
therapeutic scheme, the relatives are informed. The relatives are
also informed of the difficulties and risks of such treatment, to
be administered to such a debilitated patient. The wife decides to
refuse the conventional approach and decides to bring the husband
home and try the "harmless" therapy of baking soda, which is
administered in a lower dosage than in the preceding cases. That
restores appetite and a satisfactory digestive functionality.
For about eight months the patient has difficulty regaining
weight. After this, the improvement is more and more evident, with
the almost complete regaining of the lost weight (within 24
months) and a considerable improvement of the patient's general
condition.
Case 5: A 51-year-old patient diagnosed at the end of
1983 with bronchial carcinoma in the lower right lobe has the
diagnosis confirmed by routine oncological tests (distinctively
positive TAK but negative bronchial residue. Surgery is
proposed.The family decides to delay surgery and try the
bicarbonate treatment. Radiological examination is performed 18
months after the treatment. During these months there are no
emophtoic episodes as occurred at the beginning of the disease.
The radiological examination still indicates the presence of a
nodular mass in the lower part of the right lobe, but its
dimensions appear to be smaller and the contours of the mass more
regular.
Case 6: A 48-year-old patient with tumour in the middle
lobe of the lung that has been confirmed by all oncological
examinations is put on a waiting list for surgery at the beginning
of 1983. Incidentally, the execution modality does not seem to be
completely defined because the neoplastic mass exceeds the limits.
The patient leaves the hospital against the advice of doctors - to
the point that the doctors look for him for several months. He
then submits to a bicarbonate therapy which is able to
re-establish healthy conditions.
A radiological examination performed after nine months
reveals that the neoplastic mass has been replaced by a tenuous
transversal line located at the base of the medium lobe that can
be interpreted as a residual scar. The patient is still living.
Case 7: In 1981, a 55-year-old patient is affected by
rectal neoplasy that has been evidentiated through symptoms such
as problems with evacuation and bleeding, and, instrumentally,
through endoscopic examination. Doctors suggest rectal resection
and consequent surgical construction of a preternatural anus. In
the attempt to avoid this mutilation, the patient submits to a
local therapy with bicarbonate performed with enemas containing a
high bicarbonate solution -- 8 teaspoons per litre. Three years
after the treatment, the patient was still living.
**Critical considerations**
Having explained the theory and having briefly
illustrated the cases, it seems appropriate to analyse, in a
critical and self-critical spirit, what may emerge in neoplastic
pathology that is new and concrete.
If we closely observe the proposed therapeutic approach
it is possible to see that, independently of its real
effectiveness, it has value as an innovative theory. First, it
challenges the present methodology and especially its assumptions.
Second, it offers a concrete alternative proposal to a mountain of
conjectures and postures that sound authoritative but are too
generic and therefore ineffective.
The identification of one tumoral cause, even with all
the possible general provisos, would represent a step forward that
is indispensable for escaping that passivity determined by a lack
of results, and which is responsible for medical behaviours that
are based too much on faith and not enough on real confidence.
Given, therefore, that an unconventional medical
approach can benefit some patients better - from any point of view
- than the official treatments, and since valuable results can be
demonstrated, this should stimulate us to pursue further research
while avoiding patronising postures that are both limiting and
non-productive.
We can therefore discuss whether or not sodium
bicarbonate is the real reason for the recoveries or if, instead,
those recoveries are due to the interaction of a number of
conditions that have been created, the results of unidentified
neuro-psychical factors, or maybe the results of something totally
unknown. What is beyond question, however, is the fact that a
certain number of people, by not following conventional methods,
have been able to go back to normality without suffering and
without mutilation.
The message of this experience is therefore a call to
search for those solutions that are in accord with the simple
Hippocratic premise of man's "well-being"; that is, we must be a
stimulated to a critical evaluation of our contemporary
oncological therapies which indubitably can guarantee suffering.
One thing is certain: nowadays it is no longer
legitimate (for we are the prey of panic and of the "tumoral
syndrome"), to tolerate the slaughtering of patients in the name
of a "compassionate" obligation to help and be helped, without the
support of solid etiological foundations.
If, for a moment, we take a different point of view and
try to look at the world of the tumour with new eyes, that is, by
hypothesising a simpler genesis of neoplastic proliferation, even
the fungal one, we may be appalled and frightened by the ignorant
hand of official medicine - a hand that is armed, however, with
great cynicism and profound superficiality.
One could argue that the failures represent the
inevitable price to pay to save people's lives. But when the
suffering and the "authorised deaths" overwhelm the patient
recoveries (that seem, indeed, to be random or due to factors not
related to the therapies performed), then it is no longer
acceptable to operate at all costs and regardless of the
consequences, for in doing so, we are destined only to hurt
people.
One can rebut that the recoveries obtained by using
present oncological protocols are not so few, and that in certain
types of tumour recoveries are a high percentage. It is easy to
see, however, that these results are nothing but the outcome of
propaganda sustained by surreptitious argumentation shedding false
light on the subject of tumoral nosological entities.
When we group together both malignant tumours that are
occasionally or never healed (such as lung and stomach), tumours
that border with benignity (such as the majority of thyroid and
prostatic tumours, etc.) or put them together with those that have
an autonomous positive outcome notwithstanding chemotherapy (i.e.
infantile leukaemia) - all of this appears to be devious and
misleading, having only the purpose of forging a consensus that
would otherwise be impossible to obtain with intellectually
ethical behaviour.
If, for example, out of a certain number of tumour
species only one is susceptible to regression, it is not
legitimate to create a nosologic diagram reporting on the global
incidence of applied therapeutics regardless of the total
neoplasm's. In fact, it would be more appropriate to report the
uselessness, even the harmfulness of doing so, and leave an open
field for alternative hypotheses as far as the demonstration of
positive behaviour by the heteroplasm is concerned.
If, for example, we go back for a moment to infantile
leukaemia, the frequent positive outcomes can be correlated with
elements that are extraneous to the therapies administered. For
example, they can be correlated with those common supportive
therapies, which are considered particularly effective in young
organisms. They can be correlated with the ability of the
connective tissue to acquire, in a particular stage of growth and
development, that maturity which is necessary to the strengthening
of an immunological activity that is, at a certain point in life,
intrinsically insufficient.
It is in fact frequent in medicine that some diseases disappear
spontaneously, without apparent reason, but in correlation with
certain transitions of organic maturation.
On the oncological-mycological issue, it is known how
psoriasis and some chronic and recurrent mycoses of infancy that
reject any treatment suddenly, at a certain stage of the body's
development, disappear completely without a trace.
From the examples noted, which could be uselessly
multiplied ad infinitum, it is evident that the full panorama of
tumoral disease is extremely varied and complex. It follows that,
taking postures that are exclusive or preclusive, whether they are
conventional or unconventional, may indicate a lack of vision.
This is especially so since the terrain we are exploring is
largely unknown, and therefore cannot be charted in a way that is
uniform or standardised.
Wherever we consider an environment occupied by
invisible ultra-microscopic elements, and since the structure of
knowledge must inevitably rest on the construction of a
multiplicity of theoretical entities, there is a risk of slipping
from a perception that reflects reality to one that is merely
fictional. The acceptance of such a fictional construct may become
a pernicious reality.
The fact that modern medicine not only cannot offer sufficient
interpretative criteria but even uses dangerous methodologies that
are also harmful and meaningless - even if carried out with good
faith - is something which must push us all to search for humane
and logical alternatives. At the same time, it is necessary to
carefully, open-mindedly, and logically consider any theory or
point of view that is dared to be advanced in the battle against
that monstrous and inhuman yoke that is the tumour.
To this end, a note of acknowledgement is to go to all
those who are aware of the harmfulness of conventional therapeutic
methods and constantly try to find alternative solutions.
People like Di Bella, Govallo and others, though guilty
of utilising the same inauspicious principles of official medicine
(thus showing an excessively conformist mindset) are actually
using common sense by trying to relieve the suffering of cancer
patients through the use of painless methodologies and, in some
cases, are able to achieve remissions even though in the dark
about the real causes of cancer.
In an alternative perspective, then, it would be
necessary to conceive a new approach to experimentation in the
oncological field, setting epidemiological, etiological,
pathogenical, clinical and therapeutical research in line with a
renewed microbiology and mycology that would probably drive to the
conclusion already illustrated; that is, the tumour is a fungus -
the Candida Albicans.
The possible discovery that not only tumours but also
the majority of chronic degenerative disease could be reconciled
to mycotic causality would represent a qualitative quantum leap,
which, by revolutionising medical thinking, could greatly improve
life expectancy and quality of life. Such reconciliation might
include a wider spectrum of fungal parasites (for example, in
diseases of the connective tissues, multiple sclerosis, psoriasis,
some epileptic forms, diabetes II, etc.).
In closing, if the world of fungi - those most complex
and aggressive micro-organisms - has until now too often been
bypassed and left unobserved, the hope of this work is that we
will quickly become aware of the hazards of these micro-organisms
so that medical resources can be channelled not up blind alleys
but toward the real enemies of the human organism: external
infectious agents.
**Notes:**
1) Feyerabend P.K., "Contro il metodo", Milano 1994,
page 26
2) Verona O., "Il vasto mondo dei funghi", Bologna 1985,
page 1
3) Ibid., page2
4) Rambelli A., "Fondamenti di micologia", Bologna 1981,
page 35
5) Ibid.
6) Ibid., page 28
7) Verona O., cit. page 5
8) Rambelli A., cit. page 31
9) Ibid., page 28
10) Ibid., page 29
11) Ibid., page 266
12) Ibid., page 273
---
**Successful Treatments** -- Bladder cancer \* Brain cancer
cancer Breast Cancer \* Cancer of the spleen \* Intestinal
cancer \* Liver cancer \* Lung cancer \* Oropharynx cancer \*
Peritoneal carcinosis \* Pleura tumor \* Prostate tumor \* Skin
cancer \* Stomach cancer \* Tumor of the pancreas \* Tumors of the
limbs \* Tumor of the colon \* Prostate adenocarcinoma \* Prostate
carcinoma \* Terminal carcinoma of uterine cervix \* Peritoneal
carcinosis in adenocarcinoma of endom \* Non Hodgkin Lymphoma \*
Cerebral metastasis in diffused melanoma \* Right eye melanoma \*
Ewing's Sarcoma \* Lung cancer \* Relapsing bladder neoplasm \*
Hepatic metastases from cholangiocarcinoma \* Medullar metastatic
compression \* Hepatic carcinoma \* Hepatic carcinoma with pulmonary
metastasis \*
---
**SAFETY OF SODIUM BICARBONATE AND ITS USE IN OTHER PATHOLOGIES**
Doses of sodium bicarbonate at 5%, as indicated in the
Simoncini treatment are innocuous. In fact they have been used
without any problems for over 30 years in a multitude of other
deseases such as:
\* Severe diabetic ketoacidosis (1) \* Cardiopulmonary
resuscitation (2) \* Pregnancy (3) \* Hemodialysis (4) \* Peritoneal
dialysis. (5) \* Pharmacological toxicosis. (6) \*
Hepatopathy. (7) \* Vascular surgery operations (8)
1. Gamba, G., Bicarbonate therapy in
severe diabetic ketoacidosis. A double blind, randomized, placebo
controlled trial. (Rev Invest Clin 1991 Jul-Sep;43(3):234-8).
Miyares Gomez A. in Diabetic ketoacidosis in childhood: the first
day of treatment (An Esp Pediatr 1989 Apr;30(4):279-83).
2. Levy, M.M., An evidence-based
evaluation of the use of sodium bicarbonate during cardiopulmonary
resuscitation (Crit Care Clin 1998 Jul;14(3):457-83). Vukmir,
R.B., Sodium bicarbonate in cardiac arrest: a reappraisal (Am J
Emerg Med 1996 Mar;14(2):192-206). Bar-Joseph, G., Clinical use
of sodium bicarbonate during cardiopulmonary resuscitation--is it
used sensibly? (Resuscitation 2002 Jul;54(1):47-55).
3. Zhang. L.,Perhydrit and sodium
bicarbonate improve maternal gases and acid-base status during the
second stage of labor Department of Obstetrics and Gynecology,
Xiangya Hospital, Hunan Medical University, Changsha 410008.
Maeda, Y., Perioperative administration of bicarbonated solution
to a patient with mitochondrial encephalomyopathy (Masui 2001
Mar;50(3):299-303).
4. Avdic. E., Bicarbonate versus
acetate hemodialysis: effects on the acid-base status (Med Arh
2001;55(4):231-3).
5. Feriani, M., Randomized long-term
evaluation of bicarbonate-buffered CAPD solution. (Kidney Int
1998 Nov;54(5):1731-8).
6. Vrijlandt, P.J., Sodium
bicarbonate infusion for intoxication with tricyclic
antidepressives: recommended inspite of lack of scientific
evidence Ned Tijdschr Geneeskd 2001 Sep 1;145(35):1686-9).
Knudsen, K., Epinephrine and sodium bicarbonate independently and
additively increase survival in experimental amitriptyline
poisoning. (Crit Care Med 1997 Apr;25(4):669-74).
7. Silomon, M., Effect of sodium
bicarbonate infusion on hepatocyte Ca2+ overload during
resuscitation from hemorrhagic shock. (Resuscitation 1998
Apr;37(1):27-32). Mariano, F., Insufficient correction of blood
bicarbonate levels in biguanide lactic acidosis treated with CVVH
and bicarbonate replacement fluids (Minerva Urol Nefrol 1997
Sep;49(3):133-6).
8. Dement'eva, I.I., Calculation of
the dose of sodium bicarbonate in the treatment of metabolic
acidosis in surgery with and deep hypothermic circulatory arrest
(Anesteziol Reanimatol 1997 Sep-Oct;(5):42-4).
---
**SIMONCINI CANCER THERAPY - PROTOCOL TREATMENTS OVERVIEW**
Please be advised that for type of treatments requiring
only dropping, whashing, drinking and for psoriasis or skin cancer
the supervision of a doctor is indicated. For other type of cancer
the involvement of a doctor is mandatory.
\* 360 deg TAT (turn around treatment)
When sodium bicarbonate administered in a cavity
Lay down on the bed
2 pillows under the pelvis
Turn around 90 deg degrees every 15 minutes
Positions: supine, left and right side, prone
\* Basalioma \* Bladder cancer \* Bone cancer \* Brain
cancer \* Breast cancer \* Cervical cancer \* Choroidis melanoma \*
Conjunctive cancer \* Epithileoma \* Eye cancer \* Larinx cancer \*
Limbs cancer \* Liver cancer \* Lung cancer \* Melanoma \* Oral cancer
\* Oesophagus cancer \* Pancreas cancer \* Pediatric oncology \*
Peritoneum cancer \* Pleural cancer \* Prostate cancer \* Psoriasis \*
Rectum cancer (only when in the cavity) \* Skin cancer \* Stomach
cancer \* Uterus cancer \* Vaginal cancer
---
**SIMONCINI CANCER THERAPY - THE TREATMENT BY SODIUM BICARBONATE**
A logical solution to the cancer problem, based on the
arguments put forward so far, seems to stem from the world of
fungi against which, at the moment, there is no useful remedy
other than, in my opinion, sodium bicarbonate. The anti-fungins
that are currently on the market, in fact, do not have the ability
to penetrate the masses (except perhaps early administrations of
azoli or of amfotercina B delivered parenterally), since they are
conceived to act only at a stratified level of the epithelial
type. They are therefore unable to affect mycelial aggregations
that are set volumetrically and also when masked by the
connectival reaction that attempts to circumscribe them.
We have seen that fungi are also able to quickly mutate
their genetic structure. That means that after an initial phase of
sensitivity to fungicides, in a short time they are able to codify
them and to metabolize them without being damaged by them
rather, paradoxically, they extract a benefit from their high
toxicity on the organism.
This happens, for example, in the prostate invasive
carcinoma with congealed pelvis. There is a therapy with
anti-fungins for this affliction, which at first is very effective
at the symptomatological level but consistently loses its
effectiveness with time.
Sodium bicarbonate, instead, as it is extremely
diffusible and without that structural complexity that fungi can
easily codify, retains its ability to penetrate the masses for a
long time. This is also and especially due to the speed at which
it disintegrates them, which makes it impossible for the fungi to
adapt so that it cannot defend itself. A therapy with bicarbonate
should therefore be set up using a strong dosage, continuously,
and in cycles without pauses in a work of destruction which should
proceed from the beginning to the end without interruption for at
least 7-8 days for the first cycle, keeping in mind that a mass of
2-3-4 centimeters begins to regress consistently from the third to
the fourth day, and collapses from the fourth to the fifth.
Generally speaking, the maximum limit of the dosage that
can be administered in a session gravitates around 500 cm3 of
sodium bicarbonate at five per cent solution, with the possibility
of increasing or decreasing the dosage by 20 per cent as a
function of the body mass of the individual to be treated and in
the presence of multiple localizations upon which to apportion a
greater quantity of salts.
We must underline that the dosages indicated, as they
are harmless, are the very same that have already been utilized
without any problem for more than 30 years in a myriad of other
morbid situations such as:
\* Severe diabetic ketoacidosis [64] \* Cardio-respiratory
reanimation [65] \* Pregnancy [66] \* Hemodialysis [67] \* Peritoneal
dialysis [68] \* Pharmacological toxicosis [69] \* Hepatopathy [70]
\* Vascular surgery [71]
---
****[http://www.quackwatch.org](http://www.quackwatch.orgT)****
**[T](http://www.quackwatch.orgT)his article is a modified version of
an article originally published in Dutch on November 17,
2007 by the [Vereniging tegen
de Kwakzalverij (Dutch Association against Quackery)](http://www.kwakzalverij.nl/681/De_kankertherapie_van_Dottor_Tullio_Simoncini_uit_Rome). Dr. Koene is emeritus professor of
Nephrology at the University of Nijmegen, The Netherlands.
Ms. Josephus Jitta is assistant professor of Italian
language acquisition at the University of Amsterdam. Both
are board members of the Association.**
****Be Wary of Simoncini Cancer Therapy****
**Rob Koene, M.D.,
Ph.D.
Sophie Josephus Jitta**
Tullio
Simoncini claims that cancer is caused by a fungus and can be
cured with the administration of sodium bicarbonate. There is no
scientific evidence to support this claim, and there is good
reason to believe that the treatment is dangerous.
In October 2007, a charge was
brought against the Clinic for Preventive Medicine (CPM) in
Bilthoven, the Netherlands. This clinic houses a mixture of small
enterprises, where physicians and nonphysicians offer a great
variety of "alternative" treatments. A 50-year- old patient with
breast cancer who was treated at this clinic was admitted to the
emergency department of the University Medical Center of the Free
University of Amsterdam, where she died within a few days. The
attending physician refused to sign a death certificate, because
the patient had died from a non-natural cause. It appeared that
Simoncini had treated treated her at the Bilthoven clinic with
injections and infusions of sodium bicarbonate. The clinic medical
director denied any involvement, but two tenacious journalists of
the Dutch newspaper *de Volkskrant* succeeded in finding
out what had happened. The Public Prosecutor and the Netherlands
Health Care Inspectorate have opened an investigation of the case.
Because one of us (SJJ) is fluent in
Italian, we could extensively search Italian Web sites for
information on Simoncini's background. Currently living in Rome,
he has been using unsubstantiated cancer treatments for 15 years.
He calls himself a specialist in diabetes and metabolic diseases,
but in 2003, his license to practice medicine was withdrawn, and
in 2006 he was convicted by an Italian judge for wrongful death
and swindling. This has not stopped him from continuing to provide
his controversial treatments, not only in Italy, but apparently
also in foreign countries, such as the Netherlands. He has
appealed his conviction, but we could not find information on the
status of this appeal on Italian Web sites.
Simoncini claims that cancer is
simply an infection (*il cancro e un fungo*) caused by *Candida
albicans*, an opportunistic fungus. He claims that this
intruder causes formation of cysts and an uncontrolled cell
division in several organs, such as the liver and lungs. To
eliminate fungal colonies, he administers sodium bicarbonate by
intravenous infusion, by mouth, or even with intra-arterial
catheters close to the tumor site. Simoncini claims that the
tumors will become smaller and subsequently disappear completely
in half of patients thus treated. He does not give any proof for
this and has never published any data in a scientific journal. He
also claims that the treatment is not dangerous, because sodium
bicarbonate is also used in standard medical procedures. He fails
to mention that this treatment is applied only in patients with
definite disturbances of water and mineral metabolism and under
meticulous clinical supervision. The highly concentrated solutions
that he administers within a short period can disturb the mineral
balance in the body and lead to serious and even fatal
complications.
Based on expert reports of two
physicians, the Dutch Health Care Inspectorate has concluded that
Simoncini's treatment is dangerous and should not be administered.
Here is a translation of its news release:
**News from the
Netherlands Health Inspectorate**
February 4, 2008
**The
Administration of Sodium Bicarbonate to Cancer Patients is
Hazardous.**
The infusion of sodium bicarbonate to
vulnerable patients is hazardous and ineffective. This is the
conclusion of two expert physicians who wrote reports on request
of The Netherlands Health Inspectorate (Inspectie voor de
Gezondheidszorg, IGZ). The IGZ asked them for advice when in 2007
a patient with cancer died in the Free University Medical Centre
in Amsterdam after she had received sodium bicarbonate in a clinic
in Bilthoven. Currently, the clinic has, under the pressure of the
IGZ, stopped administering this therapy and will not restart it.
In the meantime it has not been firmly established that the
patient has died as a consequence of the sodium bicarbonate
administration. The Public Prosecutor is still investigating this.
Based on the expert report, the IGZ
has first of all reached the conclusion that there are no
scientific data that justify the administration of sodium
bicarbonate to patients with cancer for other indications than
described in the official prescription information. There is no
scientific proof whatsoever showing that this therapy cures or can
slow its progress.
The IGZ concludes that the
administration of sodium bicarbonate even has risks for patients
with high blood pressure, patients with diseases of lungs, heart,
or kidneys and for patients with cancer. This is certainly the
case if a number of specific blood levels are not monitored daily
before, during and after the treatment. The balance of the body
can become completely disturbed when large amounts are
administered. In severely ill patients, this may lead to organ
damage. In sick people, there is in fact irresponsible health care
if this product is administered without monitoring.
Given these risks and because there
is no scientific basis for the effectiveness of sodium bicarbonate
apart from the registered indications, the IGZ concludes that
physicians should not apply this treatment. If physicians
administer these despite this warning and/or the IGZ receives
reports of cases thereof, the reports will of course be
investigated, whereby the aforementioned considerations will play
an important role. The IGZ will not hesitate to inform the
Disciplinary Medical Board.
#### Additional Information
#### The Cancer Is Not a Fungus Web site claims to debunk several of Simoncini's claims --
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